Some consolidation in the area of cloud services for enterprises: Intuit, the business services company that targets SMEs, is buying Elastic Intelligence, creators of Connection Cloud, a product that helps SMEs create DIY cloud-based apps. Financial terms of the deal were not disclosed.Source: http://feedproxy.google.com/~r/Techcrunch/~3/lirPygitVH8/
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Portables aren't the only ATIV devices Samsung has in store today: Samsung's DJ Lee unveiled the ATIV One 5 Style this afternoon, an all-in-one PC with a slim 4.5mm metal frame and a Galaxy-style white bezel. From what we've seen so far, it's clearly a Windows 8 PC, and it's all screen (of the touch variety). Samsung European PC business director Patrick Povel calls it, "The perfect home entertainment device."
But you've already seen the ATIV One 5, you wanna know what's inside it -- we've just learned a load more about its specs. The ATIV One 5 features an AMD A6 quad-core processor (which also handles graphics duties), and has 4GB of RAM. The 21.5-inch display has a 1920 x 1080 resolution -- we're still not sure what type of screen that is, sadly, but we've asked. The ATIV One 5 Style arrives in the UK "later this year" for an unknown price.
Filed under: Desktops, Samsung
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BAGHDAD (AP) ? A blistering string of apparently coordinated bombings and a shooting across Iraq killed at least 40 and wounded dozens Sunday, spreading fear throughout the county in a wave of violence that is raising the prospect of a return to widespread sectarian killing a decade after a U.S.-led invasion.
Violence has spiked sharply in Iraq in recent months, with the death toll rising to levels not seen since 2008. Nearly 2,000 have been killed since the start of April, including more than 170 this month.
The surge in bloodshed accompanies rising sectarian tensions within Iraq and growing concerns that its unrest is being fanned by the Syrian civil war raging next door.
Most of Sunday's car bombs hit Shiite-majority areas and caused most of the casualties. The blasts hit half a dozen cities and towns in the south and center of the country.
There was no claim of responsibility for any of the attacks, but they bore the hallmark of al-Qaida in Iraq, which uses car bombs, suicide bombers and coordinated attacks, most aimed at security forces and members of Iraq's Shiite majority.
The U.S. Embassy condemned the attacks, saying it stands with Iraqis "who seek to live in peace and who reject cowardly acts of terrorism such as this." The U.S. withdrew its last combat troops from Iraq in December 2011, though a small number remain as an arm of the embassy to provide training and facilitate arms sales.
Sunday's blasts began with a parked car bomb exploding early in the morning in the industrial area of the city of Kut, killing six people and wounding 15 others. That was followed by another car bomb outside the city that targeted construction workers. It killed five and wounded 12, according to police.
In a teahouse hit by one of the blasts, a blood-stained tribal headdress and slippers were strewn on the floor, alongside overturned chair and couches. Kut is 160 kilometers (100 miles) southeast of Baghdad.
In the oil-rich city of Basra in southern Iraq, a car bomb exploded on a busy downtown street. As police and rescuers rushed to the scene of the initial blast, a second car exploded. Six people were reported killed. Cleaners were seen sweeping up pieces of the car bomb, which damaged nearby cars and shops.
About an hour later, parked car bombs ripped through two neighborhoods in the southern city of Nasiriyah, 320 kilometers (200 miles) southeast of Baghdad, killing two and wounding 19, police said.
In the Shiite holy city of Najaf, 160 kilometers (100 miles) south of Baghdad, a blast struck a produce market, killing eight and wounding 28. Watermelons, tomatoes and apples were seen scattered on the ground. A bulldozer loaded charred and twisted stalls and cars into a waiting truck.
Blasts were also reported in the communities of Hillah, Mahmoudiya and Madain, all south of Baghdad, killing seven in total. In the northern city of Tuz Khormato, a roadside bomb targeted a passing police patrol, killing two policemen.
The shooting broke out near the restive northern city of Mosul. Police officials say gunmen attacked police guarding a remote stretch of an oil pipeline, killing four and wounding five. Mosul, some 360 kilometers (225 miles) northwest of Baghdad, has been the scene of some of the deadliest unrest outside the Baghdad area in recent weeks.
Medical officials confirmed the casualty figures. Officials spoke on condition of anonymity as they weren't allowed to release the information to reporters.
The attacks came a day after the leader of al-Qaida's Iraq arm, known as the Islamic State of Iraq, defiantly rejected an order from the terror network's central command to stop claiming control over the organization's Syria affiliate, according to a message purportedly from him.
Abu Bakr al-Baghdadi's comments reveal his group's determination to link its own fight against the Shiite-led government in Baghdad with the cause of rebels trying to topple the Iran-backed Syrian regime.
___
Associated Press writers Sameer N. Yacoub and Adam Schreck contributed.
___
Follow Sinan Salaheddin on Twitter at twitter.com/sinansm
Source: http://news.yahoo.com/series-attacks-kill-40-people-across-iraq-161438085.html
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By Lawrence Hurley
WASHINGTON (Reuters) - The Supreme Court on Monday struck down an Arizona law that required people registering to vote in federal elections to show proof of citizenship, a victory for activists who say it discouraged Native Americans and Latinos from voting.
In a 7-2 vote, the court said the voter registration provision of the 2004 state law, known as Proposition 200, was trumped by a federal law, the 1993 National Voter Registration Act.
The state law was strongly opposed by the Mexican American Legal Defense and Education Fund (Maldef) and Indian tribes. They said it deterred legal voters who did not have the required paperwork from registering to vote.
Nina Perales, vice president of litigation at Maldef, said the ruling "sends a strong message that states cannot block their citizens from registering to vote by superimposing burdensome paperwork requirements on top of federal law."
The ruling will affect three other states - Georgia, Alabama and Kansas - that have similar laws, and prevent others that may have wanted to follow suit from enacting legislation along the same lines.
Democrats say the measures, championed by Republicans, are intended to make it more difficult for certain voters who tend to vote Democratic to cast ballots.
While issuing its ruling, the court made clear that Arizona could still have other ways to assert its argument that it should be allowed to ask for proof of citizenship. That would be the subject of separate litigation, it wrote.
Arizona Attorney General Tom Horne, who argued the state's case before the Supreme Court justices, did not immediately respond to a request for comment.
Federal law requires prospective voters to provide one of several possible forms of identification, such as a driver's license or a passport, but no proof of citizenship is needed. Would-be voters simply sign a statement saying they are citizens.
SCALIA FOR THE MAJORITY
In the Monday's majority opinion, Justice Antonin Scalia said the state law was preempted by language in the federal statute saying that states must "accept and use" a federal registration form. The state law ordered officials to reject the form if there was no accompanying proof of citizenship.
In outlining the limitations of the ruling, Scalia focused on the role of the U.S. Election Assistance Commission (EAC), a federal agency that oversees changes to state voter registration procedures.
The commission rejected the Arizona plan, prompting several justices to ponder during oral arguments in April why the state did not file a lawsuit challenging the decision.
Scalia said Arizona could still ask the commission to include a citizenship provision on the federal form in the future and could challenge the current form in separate litigation.
"That alternative means of enforcing its constitutional power to determine voting qualifications remains open to Arizona here," he wrote.
The two dissenters, Justices Clarence Thomas and Samuel Alito, said in their separate opinions that states alone have the authority to decide voter qualifications.
The case began when Arizona residents, civil rights groups and Indian tribes sued to challenge the state measure, which they said discriminated against otherwise eligible voters - among them members of more than a score of Native American tribes across the rugged desert state, some of whom struggle to meet additional requirements.
Arizona, which shares a border with Mexico, has a reputation for passing tough anti-immigration laws that have brought it into conflict with the Obama administration.
The case is Arizona v. Inter Tribal Council of Arizona, U.S. Supreme Court, No. 12-71.
(This story has been corrected to fix name spelling Pareles to Perales in fourth paragraph)
(Reporting by Lawrence Hurley and Tim Gaynor; Editing by Howard Goller and David Brunnstrom)
Source: http://news.yahoo.com/supreme-court-strikes-down-arizona-voter-registration-law-142855720.html
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Alex Cobb never lost consciousness after taking an Eric Hosmer line drive off the right side of his head on Saturday night, but he was diagnosed at Bayfront Medical Center with a mild concussion and is going to miss at least one turn in the Rays? rotation.
Joe Smith of the Tampa Bay Times reports that Cobb has been placed on the 7-day concussion disabled list. Josh Lueke was recalled from Triple-A Durham on Sunday morning to fill the vacant 25-man roster spot. He is going to pitch out of the bullpen until a fill-in for Cobb is called up later this week.
Cobb has taken a big step forward this year, boasting a 3.01 ERA, 1.16 WHIP and 76/23 K/BB ratio in 83 2/3 innings. The hope is that the 24-year-old righty will be ready to return to the mound after one week of rest.
************************
UPDATE, 3:49 PM ET:?According to Smith, Cobb has been released from Bayfront Medical Center.
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China's Xiaomi has certainly made a name for itself in the smartphone market, but let's not forget that it has other plans as well. For one, there's the Xiaomi Box, which is the company's first foray into the video content world. And according to the above leak, the next step from there appears to be a 47-inch 1080p TV, which is simply branded as "Xiaomi TV" in Chinese (model number L47M1-AA). Like the Xiaomi Box, this TV will apparently feature built-in WiFi and "MiLink" (Airplay, DLNA plus Miracast), as well as audio certification from Dolby and DTS. More after the break.
Filed under: Home Entertainment, HD
Source: Sina Weibo (login required)
Source: http://feeds.engadget.com/~r/weblogsinc/engadget/~3/xgpAz-QwI-U/
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Public release date: 2-May-2013
Share ] Contact: Vanessa Pavinato
media@esmo.org
European Society for Medical Oncology
Lugano-CH, Brussels-BE, 2 May 2013 -- A study that aimed to understand how the cancer drug everolimus helps overcome the resistance breast cancers can develop to trastuzumab has left researchers contemplating a puzzle.
The study showed a statistically non-significant benefit in clinical response rates for some patients with early breast cancer when everolimus was added to treatment with trastuzumab. Yet the results suggest this benefit is achieved independently of the molecular pathways researchers expected would be involved.
Prof Mario Campone, Principal Investigator at Institut Cancerologie de l'Ouest in Nantes, France, presented the findings at the 5th IMPAKT Breast Cancer Conference in Brussels, Belgium. The IMPAKT meeting presents cutting edge, 'translational' breast cancer research that is beginning to have an impact for patients.
"As more targeted cancer drugs are developed, the challenge is to identify which patients will benefit from individual agents," Prof Campone said. "One of the objectives of this study was to determine molecular biomarkers that predict whether a patient's cancer is sensitive to the combination of everolimus and trastuzumab compared to trastuzumab alone."
Trastuzumab is a monoclonal antibody targeted against the HER2 tyrosine kinase receptor. Many patients who initially respond to trastuzumab develop resistance.
In preclinical studies, everolimus, an oral inhibitor of an important molecule called mammalian target of rapamycin (mTOR), has demonstrated an ability to reverse trastuzumab resistance. However, the mechanisms of action involved in the reversion of trastuzumab resistance are not completely understood.
"Resistance to trastuzumab may result from several molecular alterations occurring at different levels of the downstream effectors in the PI3K/AKT pathway, all of them resulting in maintenance of signal transduction," Prof Campone explains. "Therefore, using everolimus to inhibit mTOR, a major downstream effector of this pathway, can restore sensitivity to trastuzumab. In a pre-clinical model, everolimus also reverses trastuzumab resistance caused by upregulation of IGF-1R expression, an alternative signaling pathway, allowing IGF-1 to drive cell growth and proliferation."
At the IMPAKT meeting, Prof Campone's group report the first results of a clinical study and analysis of seven biomarkers in patients with early HER2-overexpressing cancers who were receiving treatment with trastuzumab alone, or everolimus plus trastuzumab.
Among the 80 patients (40 per arm), the clinical response rate was 35% in the trastuzumab arm and 45% in the patients who received both drugs. On the other hand, the pathological response rate was 43.5% among patients in the monotherapy arm, and 47.5% in the combination arm of the study.
"The conclusion of this paper in clinical practice is that the addition of everolimus to trastuzumab seems to improve the clinical response rate but not the pathologic response," Prof Campone said.
The researchers also studied a group of seven molecular markers to explore whether they could be used to predict which patients would respond to the combination of everolimus and trastuzumab. The biomarkers were p4EBP1, pS6, eIF4E, Ki67, pAKT, LKB1, and caspase 3, all of which are involved in pathways that lead to the activation of mTOR.
"None of these biomarkers was able to predict which patients would see the benefit of the two drugs," Prof Campone said. "It appears the combination of everolimus and trastuzumab is effective independently of the activation of the PI3K/AKT/mTOR pathway and without any anti-proliferative and pro-apoptotic effect."
The study provides important confirmation of the benefit of adding everolimus in this clinical setting, comments Prof Christoph Zielinski, Chairman of the Department of Medicine I at Medical University Vienna, Austria.
"This is a rather small trial with a limited number of patients, although it quite unequivocally shows that the addition of everolimus to trastuzumab leads to an increase in clinical responses in Her-2/neu overexpressing breast cancer, as compared to trastuzumab alone," he said.
"Thus, it not only adds to clinical possibilities which can be offered to patients, but also further explains the modalities of molecularly directed therapeutic manipulation in breast cancer," said Prof Zielinski. However, the limited pathologic response constitutes a clear drawback from clinical observations.
"Nevertheless this study contributes to our assumptions on the application of molecularly targeted therapies in breast cancer in particular and in malignancies in general. It also helps to move to an era where the targeting of multiple molecular structures (like here with trastuzumab and everolimus) leads to ameliorated results by 'hitting' multiple growth-regulating mechanisms, but also avoiding treatment resistance which continues to be a major challenge in cancer treatment."
###
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Public release date: 2-May-2013 Share ] Contact: Vanessa Pavinato
media@esmo.org
European Society for Medical Oncology
Lugano-CH, Brussels-BE, 2 May 2013 -- A study that aimed to understand how the cancer drug everolimus helps overcome the resistance breast cancers can develop to trastuzumab has left researchers contemplating a puzzle.
The study showed a statistically non-significant benefit in clinical response rates for some patients with early breast cancer when everolimus was added to treatment with trastuzumab. Yet the results suggest this benefit is achieved independently of the molecular pathways researchers expected would be involved.
Prof Mario Campone, Principal Investigator at Institut Cancerologie de l'Ouest in Nantes, France, presented the findings at the 5th IMPAKT Breast Cancer Conference in Brussels, Belgium. The IMPAKT meeting presents cutting edge, 'translational' breast cancer research that is beginning to have an impact for patients.
"As more targeted cancer drugs are developed, the challenge is to identify which patients will benefit from individual agents," Prof Campone said. "One of the objectives of this study was to determine molecular biomarkers that predict whether a patient's cancer is sensitive to the combination of everolimus and trastuzumab compared to trastuzumab alone."
Trastuzumab is a monoclonal antibody targeted against the HER2 tyrosine kinase receptor. Many patients who initially respond to trastuzumab develop resistance.
In preclinical studies, everolimus, an oral inhibitor of an important molecule called mammalian target of rapamycin (mTOR), has demonstrated an ability to reverse trastuzumab resistance. However, the mechanisms of action involved in the reversion of trastuzumab resistance are not completely understood.
"Resistance to trastuzumab may result from several molecular alterations occurring at different levels of the downstream effectors in the PI3K/AKT pathway, all of them resulting in maintenance of signal transduction," Prof Campone explains. "Therefore, using everolimus to inhibit mTOR, a major downstream effector of this pathway, can restore sensitivity to trastuzumab. In a pre-clinical model, everolimus also reverses trastuzumab resistance caused by upregulation of IGF-1R expression, an alternative signaling pathway, allowing IGF-1 to drive cell growth and proliferation."
At the IMPAKT meeting, Prof Campone's group report the first results of a clinical study and analysis of seven biomarkers in patients with early HER2-overexpressing cancers who were receiving treatment with trastuzumab alone, or everolimus plus trastuzumab.
Among the 80 patients (40 per arm), the clinical response rate was 35% in the trastuzumab arm and 45% in the patients who received both drugs. On the other hand, the pathological response rate was 43.5% among patients in the monotherapy arm, and 47.5% in the combination arm of the study.
"The conclusion of this paper in clinical practice is that the addition of everolimus to trastuzumab seems to improve the clinical response rate but not the pathologic response," Prof Campone said.
The researchers also studied a group of seven molecular markers to explore whether they could be used to predict which patients would respond to the combination of everolimus and trastuzumab. The biomarkers were p4EBP1, pS6, eIF4E, Ki67, pAKT, LKB1, and caspase 3, all of which are involved in pathways that lead to the activation of mTOR.
"None of these biomarkers was able to predict which patients would see the benefit of the two drugs," Prof Campone said. "It appears the combination of everolimus and trastuzumab is effective independently of the activation of the PI3K/AKT/mTOR pathway and without any anti-proliferative and pro-apoptotic effect."
The study provides important confirmation of the benefit of adding everolimus in this clinical setting, comments Prof Christoph Zielinski, Chairman of the Department of Medicine I at Medical University Vienna, Austria.
"This is a rather small trial with a limited number of patients, although it quite unequivocally shows that the addition of everolimus to trastuzumab leads to an increase in clinical responses in Her-2/neu overexpressing breast cancer, as compared to trastuzumab alone," he said.
"Thus, it not only adds to clinical possibilities which can be offered to patients, but also further explains the modalities of molecularly directed therapeutic manipulation in breast cancer," said Prof Zielinski. However, the limited pathologic response constitutes a clear drawback from clinical observations.
"Nevertheless this study contributes to our assumptions on the application of molecularly targeted therapies in breast cancer in particular and in malignancies in general. It also helps to move to an era where the targeting of multiple molecular structures (like here with trastuzumab and everolimus) leads to ameliorated results by 'hitting' multiple growth-regulating mechanisms, but also avoiding treatment resistance which continues to be a major challenge in cancer treatment."
###
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2013-05/esfm-sce043013.php
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In between watching giraffes fight and people who can't parallel park, you'll find a new attraction on YouTube this week: live streams from members of US Congress. Today, the video-sharing website announced that all federal legislators will receive the ability to tap into advanced features on their YouTube channels, and yes, this includes the ability to pop off about the opposition party at a moment's notice. Naturally, YouTube is no stranger to public service, as it's served up similar live streams of both the DNC and RNC, the State of the Union address, and most recently, Coachella. It seems that C-SPAN's turf is safe for the time being, however, as you won't yet find live daily coverage of congressional sessions. As a consolation, you can hit the break for the giraffes... which is eerily similar to watching Congress.
[John Boehner photo credit: Gage Skidmore / Flickr]
Filed under: Internet
Via: The Next Web
Source: YouTube
Source: http://feeds.engadget.com/~r/weblogsinc/engadget/~3/NXW0PN6DLq4/
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Three of Boston bombing suspect Dzhokhar Tsarnaev's friends were arrested Wednesday and charged with covering up for him. Two told authorities they heard Tsarnaev brag about his bombmaking ability.
This courtroom sketch shows defendants Dias Kadyrbayev (l.) and Azamat Tazhayakov appearing in front of Federal Magistrate Marianne Bowler at the Moakley Federal Courthouse in Boston Wednesday. The two college friends of Boston Marathon bombing suspect Dzhokhar Tsarnaev were arrested and charged with removing a backpack containing hollowed-out fireworks from Tsarnaev's dorm room.
Jane Flavell Collins/AP
EnlargeThree men have been charged with impeding a federal investigation of the Boston Marathon bombings in allegations that would seem to blend criminality with teenage stupidity.
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Three college classmates of Boston bombing suspect Dzhokhar Tsarnaev have been charged with trying to cover up his tracks after the deadly Boston Marathon bombings. Elaine Quijano reports.After realizing that their college friend was being sought by police in connection with the April 15 attack, the three sought to discard evidence to help their friend ?avoid trouble,? according to court documents filed Wednesday.
By that time, Dzhokhar Tsarnaev was already in more than the usual amount of trouble for a 19-year-old.
Now his three friends ? Dias Kadyrbayev, Azamat Tazhayakov, and Robel Phillipos ? could also be in big trouble.
And the new criminal complaint against them also may shed new light on Mr. Tsarnaev?s interest in bombmaking. Previous news accounts have widely portrayed his older brother as the mastermind of the attacks, but the complaint says that two of the friends heard the younger Tsarnaev say, a month before the attacks, that he knew how to make bombs.
The document also quotes one of the friends saying that Tsarnaev appeared to have cut his hair short when the two saw each other on the campus the University of Massachusetts at Dartmouth on April 17, two days after the bombing. The timing of that apparent effort at a changed appearance is significant; the next day the FBI made photos of the bombing suspects public.
The criminal charges made public Wednesday do not allege that any of the three newly arrested men were involved in the bombings or knew about any plans. But these men could potentially face years in prison for obstructing justice.
Mr. Kadyrbayev and Mr. Tazhayakov, who are Kazakhstan nationals studying in the US, have been held in jail for more than a week on allegations that they violated their student visas. Now they are charged with conspiring to destroy, conceal, and cover up tangible objects belonging to the suspected bomber. That could carry a sentence of up to five years in prison and $250,000 fine.
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Apr. 24, 2013 ? The production of a certain kind of brain cell that had been considered an impediment to healing may actually be needed to staunch bleeding and promote repair after a stroke or head trauma, researchers at Duke Medicine report.
These cells, known as astrocytes, can be produced from stem cells in the brain after injury. They migrate to the site of damage where they are much more effective in promoting recovery than previously thought. This insight from studies in mice, reported online April 24, 2013, in the journal Nature, may help researchers develop treatments that foster brain repair.
"The injury recovery process is complex," said senior author Chay T. Kuo, M.D., PhD, George W. Brumley Assistant Professor of Cell Biology, Pediatrics and Neurobiology at Duke University. "There is a lot of interest in how new neurons can stimulate functional recovery, but if you make neurons without stopping the bleeding, the neurons don't even get a chance. The brain somehow knows this, so we believe that's why it produces these unique astrocytes in response to injury."
Each year, more than 1.7 million people in the United States suffer a traumatic brain injury, according to the Centers for Disease Control and Prevention. Another 795,000 people a year suffer a stroke. Few therapies are available to treat the damage that often results from such injuries.
Kuo and colleagues at Duke are interested in replacing lost neurons after a brain injury as a way to restore function. Once damaged, mature neurons cannot multiply, so most research efforts have focused on inducing brain stem cells to produce more immature neurons to replace them.
This strategy has proved difficult, because in addition to making neurons, neural stem cells also produce astrocytes and oligodendrocytes, known as glial cells. Although glial cells are important for maintaining the normal function of neurons in the brain, the increased production of astrocytes from neural stem cell has been considered an unwanted byproduct, causing more harm than good. Proliferating astrocytes secrete proteins that can induce tissue inflammation and undergo gene mutations that can lead to aggressive brain tumors.
In their study of mice, the Duke team found an unexpected insight about the astrocytes produced from stem cells after injury. Stem cells live in a special area or "niche" in the postnatal/adult brain called the subventricular zone, and churn out neurons and glia in the right proportions based on cues from the surrounding tissue.
After an injury, however, the subventricular niche pumps out more astrocytes. Significantly, the Duke team found they are different from astrocytes produced in most other regions of the brain. These cells make their way to the injured area to help make an organized scar, which stops the bleeding and allows tissue recovery.
When the generation of these astrocytes in the subventricular niche was experimentally blocked after a brain injury, hemorrhaging occurred around the injured areas and the region did not heal. Kuo said the finding was made possible by insights about astrocytes from Cagla Eroglu, PhD, whose laboratory next door to Kuo's conducts research on astrocyte interactions with neurons.
"Cagla and I started at Duke together and have known each other since our postdoctoral days," Kuo said. "To have these stem cell-made astrocytes express a unique protein that Cagla understands more than anyone else, it's just a wonderful example of scientific serendipity and collaboration."
Additionally, Kuo said first author Eric J. Benner, M.D., PhD, a former postdoctoral fellow who now has his own laboratory at Duke, provided key clinical correlations on brain injury as a physician-scientist and practicing neonatologist in the Jean and George Brumley Jr. Neonatal-Perinatal Research Institute.
"We are very excited about this innate flexibility in neural stem cell behavior to know just what to do to help the brain after injury," Kuo said. "Since bleeding in the brain after injury is a common and serious problem for patients, further research into this area may lead to effective therapies for accelerated brain recovery after injury."
In addition to Kuo, Eroglu and Benner, authors include Dominic Luciano, Rebecca Jo, Khadar Abdi, Patricia Paez-Gonzalez, Huaxin Sheng, David Warner and Chunlei Liu.
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Source: http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/genes/~3/zIrBOIqCR0I/130424132707.htm
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The Infinity Cell is a kinetic charger for the iPhone that uses your body's movement to generate electricity. The current prototype for the Infinity Cell is a crude 3D printed rectangle, roughly the size of a pack of cigarettes, linked up to the iPhone with a cable. The plan is to create a more streamlined version during the product's Kickstarter campaign.Source: http://feedproxy.google.com/~r/Techcrunch/~3/hPfrNCPcDNo/
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This is the story of a little smartphone series that grows up and, three years later, positions itself to take over the world. The device in question, the newest addition to that lineup, doesn't really need much of an introduction thanks to some of the most successful marketing campaigns in the world. We'll be happy to give it anyway: pictured atop this very text sits the Samsung Galaxy S 4, the latest and greatest flagship out of Korea. This is the hero, the device chosen to lead the charge for Samsung as it ventures deeper into 2013, and it's fitted with the best of everything: a 1080p Super AMOLED display, 1.9GHz quad-core (or 1.6GHz Exynos 5 Octa 5410, depending on market) chipset, Android 4.2, 13MP camera and a wide assortment of brand-new firmware amenities, to name just a few.
Despite the fact that its predecessor sold millions upon millions of units in the past year, the Galaxy S 4 isn't alone in its quest for global Android domination this time. HTC, the underdog of the fight, has launched the One, a flagship that rivals the GS 4 in almost every way and does so in a physically attractive package complete with a solid aluminum build. Where do these two devices stand in comparison to each other? Does the GS 4 reign supreme? Will its onslaught of new software features send the phone to the top of the pack? These answers and more await you after the break.
Filed under: Cellphones, Mobile, Samsung
Source: http://feeds.engadget.com/~r/weblogsinc/engadget/~3/scilblOQDl8/
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White House press secretary Jay Carney speaks during his daily news briefing at the White House in Washington, Wednesday, April, 24, 2013. (AP Photo/Pablo Martinez Monsivais)
White House press secretary Jay Carney speaks during his daily news briefing at the White House in Washington, Wednesday, April, 24, 2013. (AP Photo/Pablo Martinez Monsivais)
WASHINGTON (AP) ? The White House now says it's willing to consider legislation that would give the administration the budget-cutting flexibility to avoid furloughs of air traffic controllers.
Those furloughs could result in widespread air traffic delays.
White House spokesman Jay Carney says that if Congress wants to address the effect of automatic spending cuts on the Federal Aviation Administration, "we would be open to looking at that."
But he says that would be a "Band-Aid measure" that would not address other automatic cuts that kicked in March 1.
Back in February, when those cuts were approaching, the White House threatened to veto legislation that would give Obama greater budget-cutting flexibility.
At the time, the White House said it couldn't make dramatic cuts without affecting national security or hurting the economy.
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Feb. 27, 2013 ? Bearded capuchin monkeys deliberately place palm nuts in a stable position on a surface before trying to crack them open, revealing their capacity to use tactile information to improve tool use.
The results are published February 27 in the open access journal PLOS ONE by Dorothy Fragaszy and colleagues from the University of Georgia.
The researchers analyzed the monkeys' tool-use skills by videotaping adult monkeys cracking palm nuts on a surface they used frequently for the purpose. They found that monkeys positioned the nuts flat side down more frequently than expected by random chance. When placing the nuts, the monkeys knocked the nuts on the surface a few times before releasing them, after which the nuts very rarely moved.
The researchers suggest that the monkeys may have learned to optimize this tool-use strategy by repeatedly knocking the nut to achieve the stable position prior to cracking it. They conclude that the monkeys' strategic placement of the nut reveals that the monkeys pay attention to the fit between the nut and the surface each time they place the nut, and adjust their actions accordingly.
In a parallel experiment, the scientists asked blindfolded people to perform the same action, positioning palm nuts on an anvil as if to crack them with a stone or hammer. Like the monkeys, the human participants also followed tactile cues to place the nut flat-side down on the anvil.
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Washington and Washington State are each sending the same number of football players to the NFL Combine this week as Eastern Washington.
Desmond Trufant. (Getty Images)
One.
What does that say about the state of the UW and WSU football programs? Well, it ain?t good. We know Wazzu floundered under ex-coach Paul Wulff, but this year?s seniors were the first recruiting class for UW head coach Steve Sarkisian. Hmm.
Anyway, the only Husky headed to Indianapolis to display their talents for NFL scouts is cornerback Desmond Trufant, brother of pro corners Marcus Trufant (who went to WSU and plays for the Seahawks) and Isaiah Trufant (who went to Eastern and plays for the Jets). Desmond, the youngest Trufant brother, is declaring for this April?s NFL Draft after finishing his senior year at Washington.
Trufant had 36 total tackles, one sack, one interception and nine passes defended during the 2012 season ? arguably his worst numbers in four years as a Husky, but play that nevertheless earned him All-Pac-12 first team honors. His best season stats-wise was 2011, when he made 64 tackles, grabbed two picks, defended 16 passes and forced two fumbles.
The Tacoma native is expected to go early in the second round of the NFL Draft. The league?s official Combine website, which rated him 84.1 out of 100, had this to say:
Strengths
Legacy pick with NFL size and athleticism. Very good short-area foot quickness, mirrors receivers on the outside on double moves up the sideline or quick out routes. Capable of playing either outside or in the slot. Has speed to run with better receivers. Plays a lot of press-bail but has length and tenacity in coverage. Regularly rips off and out-quicks receiver blocks to get into position to make plays. Competes for the ball in the air and won?t back down from physical challenges from receivers.Weaknesses
Only adequate in his overall strength and physicality, often whiffs at tackle attempts, jumping at feet or throwing a weak shoulder. Has mental lapses in coverage that lead to big plays. Poor technique in press makes him susceptible to giving up plays. Gives up the sideline in the run game when failing to get outside leverage against receiver blocks. Will open up his hips far too quickly and immediately give up inside leverage.
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Marquess Wilson. (WSU Athletics photo)
For the Cougars, wide receiver Marquess Wilson is the only player headed to Lucas Oil Stadium for the NFL Combine, which runs Wednesday through next Tuesday. He is declaring for the NFL Draft after his junior year at WSU, having finished with three school records: 3,207 career receiving yards, 14 100-yard games and four 150-yard games.
But that?s not the whole story. After nine games this past season, Wilson quit the team in November and alleged misconduct by new head coach Mike Leach?s staff. In an open letter to Cougars fans Nov. 10, Wilson said the staff practiced ?physical, emotional and verbal abuse? against their players.
Wilson quickly retracted those allegations to WSU athletic director Bill Moos, and after internal and NCAA investigations the university was cleared of wrongdoing. Now, Wilson?s indefinite suspension from the team ? when he reportedly walked out of a conditioning workout and quit ? is a considerable pock-mark on him for the NFL Draft.
NFL.com gave Wilson a score of 67.3 and expects him to be drafted. ?If teams believe he can improve the consistency of his hands and mature physically and mentally at the next level,? the NFL Combine website states, ?he could be picked in the middle rounds and contribute immediately, similarly to how Mike Williams did for Tampa Bay in 2010.?
Here?s what NFL.com had to say about his play:
Strengths
Tall strider presenting a difficult ask down the sideline for shorter defensive backs. If able to escape the jam or come off the line untouched, he covers a lot of ground in a hurry. His height and vertical jump makes him a nightmare on jump balls anywhere on the field, and he can contort his body to adjust to passes in the air. Makes catches over either shoulder down the sideline and can stay in-bounds. Drops his hips, has quick enough feet and a solid head-fake to separate at the top of the route, as well as to pull off double-moves downfield and to the sideline. Tries to run through tackles by lowering his pads and churning his feet after the catch and has a bit of open-field elusiveness, though he will run backwards and dance too much instead of taking what he can get. Will find a soft spot in the zone if his quarterback is on the run. Flashes an effective use of his length, and a bit of attitude, to take smaller defenders out in the run game.Weaknesses
Lanky receiver, especially thin in the lower body. Will struggle with the physicality of NFL defenders off the line, when trying to get into his route, and downfield in 50/50 situations. Does not consistently use his hands to win or hold onto the ball when being hit or even feeling the hit coming. Hands reliably is severely lacking, misses on all difficulty of catches due to a lack of concentration. Doesn?t adjust to low passes as well as he does high ones. Lateral quickness against press is only average, takes time to accelerate into his route if challenged at all. Lacks technique as a blocker, when he makes the effort to reach his target, tries to overwhelm corners but fails to move his feet or maintain the angle to prevent them from entering the play.
?
Brandon Kaufman. (EWU Athletics photo)
Finally, there?s the lone Eastern Washington representative, wide receiver Brandon Kaufman. He stands 6-foot-5, and after red-shirting in his junior year for wrist and knee injuries, came back for 2012 and led the team with 16 touchdown receptions, 1,850 receiving yards and 132.1 yards per game.
Kaufman shined from his freshman season on, twice earning Football Championship Subdivision All-America honors and, in 2012, helping guide the Eagles to the Big Sky championship and FCS playoffs. He is just the seventh EWU player to be invited to the NFL Combine in the past 11 years.
NFL.com, which rated him 68.0 and expects him to be drafted in the middle rounds, had this to say about Kaufman:
Strengths
Tall with long arms. Uses his size to shield defenders, and his length to catch passes off his frame. Reliable hands. Tracks the football well and attacks the ball at the highest point. Red zone and vertical weapon. Possesses good vision after the catch.Weaknesses
Hasn?t dealt with great competition, or run a myriad of different routes. Doesn?t explode off the line, and has build up speed. Needs to be more physical in his route running. Might struggle to consistently separate at the next level.
Good luck, boys!
Visit seattlepi.com for more Seattle news. Contact sports editor Nick Eaton at nickeaton@seattlepi.com or @njeaton.
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The following guest post comes from Monica Corton of indie publisher Next Decade Entertainment. ? ? ?
?
I recently read an exchange on Forbes.com involving the Consumer Electronics Association?s (CEA) leader Gary Shapiro, who asserted music publishers (through our trade organization, NMPA) are working to convince Congress that our issues are, "more important than the interests of artists, musicians, innovators and ordinary people."
David Israelite (President of the NMPA) set the record straight, but I feel the need to remind everyone that songwriters and many music publishers are indeed small businesses run by "ordinary people."
?
Next Decade Entertainment, as a music publisher, represents songwriters... the people that actually write the songs you know and love. We have three full time employees, a handful of consultants and interns.
We are not to be confused with the record companies who represent the artists who sing the fabulous songs that are written by our songwriters. People often confuse these two very separate rights... the song vs. the artist's recording of the song.
I explain this distinction because music publishers, unlike record companies, are subject to government oversight when it comes to making licenses, specifically mechanical licenses (audio-only uses like a CD, digital download, streaming uses) with tech companies (Apple, Amazon, Google, etc.) and others. That's right, the government tells us what we can be paid for all mechanical licenses.
?
?
Every five years, the NMPA negotiates on behalf of music publishers and our songwriting partners through the Copyright Royalty Board (CRB), the government oversight group, to fight for that mechanical rate.
The explosive growth of internet-based music platforms has significantly intensified our CRB efforts and brought an army of groups wanting to pay songwriters less (Apple, Amazon, Google, etc.). ?The internet has also led an enormous amount of people to download music for free (often illegally). ?
?
As the consumer market shifts from buying physical product and digital downloads to streaming music, songwriters and music publishers? income has done a precipitous deep dive. Like most independent music publishers, we are successful but struggling under the weight of the attack by those who oppose us.
If you are starting to see the "David vs. Goliath" story here... you are correct.
The music publishing industry goes in front of this CRB panel as one entity advocating for a fair mechanical rate on behalf of thousands of songwriters and publishers against a massive army of corporations bent on making sure it is as low as possible.
Yet, some portray music publishers as over-reaching (just consider Pandora's constant complaint that publishers charge to much). ?
?
We, independent music publishers and songwriters, are people who need to feed our families, finance a home and send our kids to college; ordinary people trying to make a living. At the end of the day, we love music. ?We want to enable songwriters to provide the world with the best music. In all the debating, one simple fact is being forgotten. ?Songwriters are the backbone of the music
business. There is NO recording by an artist until a songwriter has written a hit song to make their voice shine.
It's time to recognize the importance and value of small businesses ? Next Decade Entertainment and hundreds of others like us. ?Yes, our livelihood depends on it, but so does the fate of American music.
?
Source: http://www.digitalmusicnews.com/permalink/2013/201302publisher
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Feb. 22, 2013 ? The fruit fly study adds to the evidence "that using toxins in the environment to medicate offspring may be common across the animal kingdom," says biologist Todd Schlenke.
When fruit flies sense parasitic wasps in their environment, they lay their eggs in an alcohol-soaked environment, essentially forcing their larvae to consume booze as a drug to combat the deadly wasps.
The discovery by biologists at Emory University is being published in the journal Science on February 22.
"The adult flies actually anticipate an infection risk to their children, and then they medicate them by depositing them in alcohol," says Todd Schlenke, the evolutionary geneticist whose lab did the research. "We found that this medicating behavior was shared by diverse fly species, adding to the evidence that using toxins in the environment to medicate offspring may be common across the animal kingdom."
Adult fruit flies detect the wasps by sight, and appear to have much better vision than previously realized, he adds. "Our data indicate that the flies can visually distinguish the relatively small morphological differences between male and female wasps, and between different species of wasps."
The experiments were led by Balint Zacsoh, who recently graduated from Emory with a degree in biology and still works in the Schlenke lab. The team also included Emory graduate student Zachary Lynch and postdoc Nathan Mortimer.
The larvae of the common fruit fly, Drosophila melanogaster, eat the rot, or fungi and bacteria, that grows on overripe, fermenting fruit. They have evolved a certain amount of resistance to the toxic effects of the alcohol levels in their natural habitat, which can range up to 15 percent.
Tiny, endoparasitoid wasps are major killers of fruit flies. The wasps inject their eggs inside the fruit fly larvae, along with venom that aims to suppress their hosts' cellular immune response. If the flies fail to kill the wasp egg, a wasp larva hatches inside the fruit fly larva and begins to eat its host from the inside out.
Last year, the Schlenke lab published a study showing how fruit fly larvae infected with wasps prefer to eat food high in alcohol. This behavior greatly improves the survival rate of the fruit flies because they have evolved high tolerance of the toxic effects of the alcohol, but the wasps have not.
"The fruit fly larvae raise their blood alcohol levels, so that the wasps living in their blood will suffer," Schlenke says. "When you think of an immune system, you usually think of blood cells and immune proteins, but behavior can also be a big part of an organism's immune defense."
For the latest study, the researchers asked whether the fruit fly parents could sense when their children were at risk for infection, and whether they then sought out alcohol to prophylactically medicate them.
Adult female fruit flies were released in one mesh cage with parasitic wasps and another mesh cage with no wasps. Both cages had two petri dishes containing yeast, the nourishment for lab-raised fruit flies and their larvae. The yeast in one of the petri dishes was mixed with 6 percent alcohol, while the yeast in the other dish was alcohol free. After 24 hours, the petri dishes were removed and the researchers counted the eggs that the fruit flies had laid.
The results were dramatic. In the mesh cage with parasitic wasps, 90 percent of the eggs laid were in the dish containing alcohol. In the cage with no wasps, only 40 percent of the eggs were in the alcohol dish.
"The fruit flies clearly change their reproductive behavior when the wasps are present," Schlenke says. "The alcohol is slightly toxic to the fruit flies as well, but the wasps are a bigger danger than the alcohol."
The fly strains used in the experiments have been bred in the lab for decades. "The flies that we work with have not seen wasps in their lives before, and neither have their ancestors going back hundreds of generations," Schlenke says. "And yet, the flies still recognize these wasps as a danger when they are put in a cage with them."
Further experiments showed that the flies are extremely discerning about differences in the wasps. They preferred to lay their eggs in alcohol when female wasps were present, but not if only male wasps were in the cage.
Theorizing that the flies were reacting to pheromones, the researchers conducted experiments using two groups of mutated fruit flies. One group lacked the ability to smell, and another group lacked sight. The flies unable to smell, however, still preferred to lay their eggs in alcohol when female wasps were present. The blind flies did not make the distinction, choosing the non-alcohol food for their offspring, even in the presence of female wasps.
"This result was a surprise to me," Schlenke says. "I thought the flies were probably using olfaction to sense the female wasps. The small, compound eyes of flies are believed to be more geared to detecting motion than high-resolution images."
The only obvious visual differences between the female and male wasps, he adds, is that the males have longer antennae, slightly smaller bodies, and lack an ovipositor.
Further experimentation showed that the fruit flies can distinguish different species of wasps, and will only choose the alcohol food in response to wasp species that infect larvae, not fly pupae. "Fly larvae usually leave the food before they pupate," Schlenke explains, "so there is likely little benefit to laying eggs at alcoholic sites when pupal parasites are present."
The researchers also connected the exposure to female parasitic wasps to changes in a fruit fly neuropeptide.
Stress, and the resulting reduced level of neuropeptide F, or NPF, has previously been associated with alcohol-seeking behavior in fruit flies. Similarly, levels of a homologous neuropeptide in humans, NPY, is associated with alcoholism.
"We found that when a fruit fly is exposed to female parasitic wasps, this exposure reduces the level of NPF in the fly brain, causing the fly to seek out alcoholic sites for oviposition," Schlenke says. "Furthermore, the alcohol-seeking behavior appears to remain for the duration of the fly's life, even when the parasitic wasps are no longer present, an example of long-term memory."
Finally, Drosophila melanogaster is not unique in using this offspring medication behavior. "We tested a number of fly species," Schlenke says, "and found that each fly species that uses rotting fruit for food mounts this immune behavior against parasitic wasps. Medication may be far more common in nature than we previously thought."
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The above story is reprinted from materials provided by Emory Health Sciences. The original article was written by Carol Clark.
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Source: http://feeds.sciencedaily.com/~r/sciencedaily/~3/553YyOM3vUk/130222102958.htm
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